Biology is designed for multi-semester biology courses for science majors. It is …
Biology is designed for multi-semester biology courses for science majors. It is grounded on an evolutionary basis and includes exciting features that highlight careers in the biological sciences and everyday applications of the concepts at hand. To meet the needs of today’s instructors and students, some content has been strategically condensed while maintaining the overall scope and coverage of traditional texts for this course. Instructors can customize the book, adapting it to the approach that works best in their classroom. Biology also includes an innovative art program that incorporates critical thinking and clicker questions to help students understand—and apply—key concepts.
By the end of this section, you will be able to:Describe the …
By the end of this section, you will be able to:Describe the structural organization of nematodesUnderstand the importance of Caenorhabditis elegans in researchCompare the internal systems and appendage specializations of phylum ArthropodaDiscuss the environmental importance of arthropodsDiscuss the reasons for arthropod success and abundance
This resource is a video abstract of a research paper created by …
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:
"Apoptosis, a form of programmed cell death, plays critical roles in animal development and in repair of DNA damage. Since DNA damage is a major factor in cancer development, identifying the regulators of damage-induced apoptosis could help researchers develop treatments. A recent study investigated whether NHR-14, an important developmental protein in the model organism C. elegans, also contributes to damage-induced apoptosis . using mutant C. elegans that are especially susceptible to radiation-induced DNA damage. Deletion of the gene encoding NHR-14, which corresponds to HNF4 in humans, decreased radiation-induced apoptosis of sex cells without affecting the levels of normal (non-damage-induced) apoptosis, indicating a specific role in the damage-induced death pathway. Further exploration revealed that the NHR-14 gene acts “downstream” of the DNA damage checkpoint pathway and regulates the transcription of the genes egl-1 and ced-13 after DNA is damaged..."
The rest of the transcript, along with a link to the research itself, is available on the resource itself.
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